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J Clin Pathol 58:166-171 doi:10.1136/jcp.2004.020909
  • Original article

Clinicopathological study of cellular proliferation and invasion in gliomatosis cerebri: important role of neural cell adhesion molecule L1 in tumour invasion

Table 1

 Patient details, histology, and immunohistochemical results

Case Age Sex Histology GFAP L1 Ki-67 (%)
Immunohistochemical analysis of 4 patients with gliomatosis cerebri and 20 patients with other gliomas.
As, astrocytoma; GFAP, glial fibrillary acidic protein; –, negative; ±, trace/positive; +, positive; ++, strongly positive.
1 47 F Gliomatosis cerebri (As-grade 3) ++ ++ 4
2 58 M Gliomatosis cerebri (As-grade 3) ++ ++ 17
3 31 M Gliomatosis cerebri (As-grade 3) ++ ++ 11
4 45 F Gliomatosis cerebri (As-grade 2) ++ ++ 5
5 67 F Pilocytic astrocytoma ++ + 4
6 20 M Fibrillary astrocytoma ++ + 2
7 52 M Fibrillary astrocytoma + ± 2
8 52 M Fibrillary astrocytoma ++ ± 4
9 43 F Gemistocytic astrocytoma ++ ± 3
10 52 F Gemistocytic astrocytoma ++ + 2
11 25 M Oligoastrocytoma ± ± 8
12 31 M Oligoastrocytoma ± ± 1
13 40 M Oligoastrocytoma ± 2
14 40 F Oligoastrocytoma ± ± 5
15 26 F Anaplastic astrocytoma ++ + 8
16 54 M Anaplastic astrocytoma ++ 5
17 50 M Anaplastic oligoastrocytoma + 19
18 27 M Glioblastoma + ± 20
19 48 F Glioblastoma ± 11
20 59 M Glioblastoma + ± 12
21 62 M Glioblastoma + 8
22 63 F Glioblastoma + 9
23 42 F Glioblastoma ± ± 12
24 58 M Glioblastoma ± ± 8

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